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(2022) [139] remind us that since oxidative damages during prostate carcinogenesis may contribute to modifications of macromolecules that trigger cellular malignant transformations, it is worth considering that functional single-nucleotide polymorphisms of enzymes engaged in redox homeostasis can also disrupt the pro-oxidant–antioxidant balance, resulting in accumulation of reactive oxygen species and oxidative damage

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“The Feds don’t know what to do,” a representative told me

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