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The fact that the glyoxalase mutants grew normally in broth but had a defect for growth in TLR-activated macrophages and in mice, suggests that host-derived MG imposes the major threat to bacteria during its intracellular growth, although we cannot rule out that changes in L

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However, this evidence is not direct, in the context of testosterone synthesis, oxidative stress particularly affects the Leydig cells in the testes, which are responsible for producing testosterone [66,67]

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